Download Neurology Volume 72(3) January 20, 2009 by American Academy of Neurology PDF

By American Academy of Neurology

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Extra resources for Neurology Volume 72(3) January 20, 2009

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7,17 Of the 48 children with ON in the current study, 40% were reported to have an abnormal MRI and thus represent a subgroup considered to be at particularly high risk for a future diagnosis of MS. Another important potential influence on clinical presentation is age at ADS onset. Of the 219 children, 95 were younger than 10 years. As expected, an ADEM presentation was particularly prominent in this age group (37%). Whether the polylesional features, global impairment of alertness (encephalopathy), and widespread MRI lesion pattern characteristic of ADEM delineate a unique pathobiology or an age-related propensity for heightened inflammation is an important area for future research.

Sensory nerve conduction velocities were antidromic. Compound muscle action potential and sensory nerve action potential amplitudes were also recorded. Temperature was maintained at 34°C in the hands and feet for all visits. Surface electrodes were used in all studies. Microsatellite analysis. Microsatellite markers DXS991, DXS1275, DXS8052, DXS559, DXS8046, DXS986, DXS6793, DXS995, DXS8076, DXS8114, DXS6803, DXS1196, DXS1217, and DXS990 were genotyped in the family. The forward primers were 5= labeled with the 6-FAM fluorochrome.

10 Families with dHMN have also shown overlap with clinical signs and pathology similar to amyotrophic lateral sclerosis (ALS) and CMT neuropathy. 7 It is likely that identifying new genes involved in dHMN will be helpful in identifying additional pathways relevant to other motor neuron disorders as well as motor neuronal function. This study reports a family with X-linked recessive adult-onset distal hereditary motor neuropathy. 13 METHODS Family ascertainment. We examined a threegeneration family with probable X-lined adult-onset hereditary motor neuron disease.

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