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By Jean Schoenen, David W. Dodick, Peter S?ndor

This e-book provides a clinically orientated method of figuring out and treating co-morbid migraine. Case vignettes and administration algorithms will increase the medical application of the e-book.

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Migraine and non-migrainous headaches. A community survey in Jerusalem. J Epidemiol Commun H 1980;34:188–93. 21 Abramson JH, Gofin J, Peritz E, Hopp C, Epstein LM. Clustering of chronic disorders – a community study of coprevalence in Jerusalem. J Chronic Dis 1982;35:221–30. 22 Bigal ME, Liberman JN, Lipton RB. Obesity and migraine: A population study. Neurology 2006;66:545–50. 23 Carson AP, Rose KM, Sanford CP, et al. Lifetime prevalence of migraine and other headaches lasting 4 or more hours: the Atherosclerosis Risk in Communities (ARIC) study.

42 Pavlakis SG, Phillips PC, DiMauro S, De Vivo DC, Rowland LP. Mitochondrial myopathy, encephalopathy, lactic acidosis, and strokelike episodes: a distinctive clinical syndrome. Ann Neurol 1984;16:481–8. 43 Terwindt GM, Haan J, Ophoff RA, et al. Clinical and genetic analysis of a large Dutch family with autosomal dominant vascular retinopathy, migraine and Raynaud’s phenomenon. Brain 1998;121:303–16. 44 Olesen J, Friberg L, Olsen TS, et al. Ischaemia-induced (symptomatic) migraine attacks may be more frequent than migraine-induced ischaemic insults.

Such studies can address, however, whether the use of triptans (and ergotamine) in practice is associated with risk of cardiovascular outcomes. Two such studies [18,19] based on managed care samples performed this analysis and are summarized below. Migraine sufferers were identified through medical records based on diagnostic history and/or prescription of migraine-specific medication. Various cardiovascular outcomes were assessed through diagnostic codes as detailed below. Data were analyzed to determine whether migraineurs thus identified were at increased risk of incident cardiovascular disease compared to non-migraineurs.

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